Molecule of the month: miRNA and Human Prion brain disease

نویسنده

  • Paul Shapshak
چکیده

Human prion brain disease has been studied intensely since 1920. Examples of such diseases include Creutzfeldt-Jakob disease (CJD), fatal familial insomnia, Gerstmann-StrausslerScheinker disease, and Kuru. Central in the epidemiology and pathogenesis of prion diseases is the prion protein itself and the gene for this protein resides on chromosome 12, at locus 20pterp12. The prion protein is termed PRNP, has a monomer molecular weight of 22-36 Kd and is linked to glycosylphosphatidyl-inositol (GPI) on cell surfaces. Various mutants of PRNP have roles in disease pathogenesis. Interestingly, the functions of the normal prion protein include synaptic interactions, cell adhesion, and transmembrane signaling [1].

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عنوان ژورنال:

دوره 9  شماره 

صفحات  -

تاریخ انتشار 2013